Acromioclavicular Joint Injury

Background

Injuries in and around the shoulder are common in today's athletic society. Proper knowledge of the different problems and treatment options for shoulder disorders is necessary to get patients back to their preinjury state.

Acromioclavicular (AC) joint injuries are common and often seen after bicycle wrecks, contact sports, and car accidents. The acromioclavicular joint is located at the top of the shoulder where the acromion process and the clavicle meet to form a joint. Several ligaments surround this joint, and depending on the severity of the injury, a person may tear one or all of the ligaments. Torn ligaments lead to acromioclavicular joint sprains and separations.[1]

The distal clavicle and acromion process can also be fractured. Injury to the acromioclavicular joint may injure the cartilage within the joint and can later cause arthritis of the acromioclavicular joint.

This article discusses the anatomy of the acromioclavicular joint, the diagnosis of disorders of this joint, and the different treatment options.

For excellent patient education resources, see eMedicineHealth's First Aid & Injuries Center. Also, see eMedicineHealth's article on Shoulder Dislocation.

NextEpidemiologyFrequencyUnited States

Injuries to the acromioclavicular joint are the most common reason that athletes seek medical attention following an acute shoulder injury. Glenohumeral dislocations (see Shoulder Dislocation) are the second most common injuries seen. Men in their second through fourth decades of life have the greatest frequency of acromioclavicular joint injuries, which are most often incomplete tears of the ligaments.[1]

PreviousNextFunctional Anatomy

The normal width of the acromioclavicula joint is 1-3 mm in younger individuals; it narrows to 0.5 mm or less in individuals older than 60 years.

The acromioclavicular joint is made up of 2 bones (the clavicle and the acromion), 4 ligaments, and a meniscus inside the joint.

The acromioclavicular joint is surrounded by a thin joint capsule and 4 small ligaments. These ligaments mostly give joint stability to anterior and posterior translation, as well as provide horizontal stability to the joint.

Another set of ligaments also provides vertical stability to the acromioclavicular joint. These ligaments are called the coracoclavicular ligaments, which are found medial to the acromioclavicular joint and go from the coracoid process on the scapula to the clavicle.

Different injuries result in different tears of the 2 coracoclavicular ligaments (the conoid and the trapezoid). Torn acromioclavicular joint ligaments and/or torn coracoclavicular ligaments are seen in acromioclavicular joint sprains. The meniscus that lies in the joint may also be injured during sprains or fractures around the acromioclavicular joint. The acromioclavicular capsular ligaments provide most of the joint stability in the anteroposterior (AP) direction. The conoid and trapezoid ligaments aid in providing superior-inferior stability to the joint. Compression of the joint is restrained mainly by the trapezoid ligament.

PreviousNextSport-Specific Biomechanics

When a person falls onto their shoulder, the force pushes the tip of the shoulder down. The clavicle is usually kept in its anatomic position, whereas the shoulder is driven down, which injures the different ligaments or causes a fracture. When the ligaments are injured they are either sprained or, in more severe cases, torn.

Acromioclavicular joint sprains have been classified according to their severity. In a type I sprain, a mild force applied to these ligaments does not tear them. The injury simply results in a sprain, which hurts, but the shoulder does not show any gross evidence of an acromioclavicular joint dislocation. Type II sprains are seen when a heavier force is applied to the shoulder, disrupting the acromioclavicular ligaments but leaving the coracoclavicular ligaments intact. When these injuries occur, the lateral clavicle becomes a little more prominent.

In type III sprains, the force completely disrupts the acromioclavicular and coracoclavicular ligaments. This leads to complete separation of the clavicle and obvious changes in appearance. The lateral clavicle is very prominent. A few more types of acromioclavicular joint sprains have been classified, but types I–III are the most common (see below).

Classification of acromioclavicular joint injuries.

An acromioclavicular joint sprain is more common than a fracture after an injury. However, fractures of the distal clavicle and the acromion process may occur, so the healthcare provider must be aware of such injuries and ready to diagnose and treat them as well (see Clavicular Injuries).

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Sacroiliac Joint Injury

Background

Lower back pain is one of the most prevalent sports maladies, affecting athletes in nearly every sport. Diagnosing the cause of a back injury is quite difficult and challenging because multiple structures in the lower back region can cause pain. However, an accurate diagnosis is paramount to providing successful treatment of the spine injury.

Although still somewhat controversial, the sacroiliac joint (SIJ) is generally accepted as an anatomic structure within the lumbar complex that if injured can be a cause of lower back pain. Mechanical dysfunction, inflammation, infection, trauma, and degeneration all have been attributed to the SIJ. Once the diagnosis of SIJ injury is established, specifically directed treatment can lead to satisfying results. This article discusses the diagnosis, management, and rehabilitation of sacroiliac injuries and pain.

For excellent patient education resources, visit eMedicineHealth's Osteoporosis Center. Also, see eMedicineHealth's patient education articles Low Back Pain and Lumbar Disc Disease.

NextEpidemiologyFrequencyUnited States

The incidence of lower back pain in humans parallels the incidence of the common cold, with a lifetime rate approaching 95%. Goldwaith and Osgood first discussed the possibility that SIJ injury could cause low back pain as early as 1905.[1] In the decades since then, several attempts have been made to establish the prevalence of SIJ syndrome in persons with back pain, and the results of these reports vary widely.

Schwarzer et al remarked that "the prevalence of sacroiliac pain would appear to be at least 13% and perhaps as high a 30%" in patients with low back and buttock pain.[2] Bernard and Kirkaldy-Willis reported the prevalence rate to be 22.5% in 1293 patients with back pain.[3]

PreviousNextFunctional Anatomy

The SIJ is a true diarthrodial joint that joins the sacrum to the pelvis.[4, 5, 6] In this joint, hyaline cartilage on the sacral side moves against fibrocartilage on the iliac side. The joint is generally C shaped with 2 lever arms that interlock at the second sacral level. The joint contains numerous ridges and depressions, indicating its function for stability more than motion. However, studies have documented that motion does occur at the joint; therefore, slightly subluxed and even locked positions can occur.[2, 7]

Stability is provided by the ridges present in the joint and by the presence of generously sized ligaments. The ligamentous structures offer resistance to shear and loading. The deep anterior, posterior, and interosseous ligaments resist the load of the sacrum relative to the ilium. More superficial ligaments (eg, sacrotuberous ligament) react to dynamic motions (eg, straight-leg raising during physical motion). The long dorsal sacroiliac ligament can become stretched in periods of reduced lumbar lordosis (eg, pregnancy).

Many large and small muscles have relationships with these ligaments and the SIJ, including the piriformis, biceps femoris, gluteus maximus and minimus, erector spinae, latissimus dorsi, thoracolumbar fascia, and iliacus. Any of these muscles can be involved with a painful SIJ. As a true joint, the SIJ is a pain-sensitive structure richly innervated by a combination of unmyelinated free nerve endings and the posterior primary rami of L2-S3. The wide possibility of innervation may explain why pain emanation from the joint can manifest in so many various ways, with different and unique referral patterns for individual patients.

PreviousNextSport-Specific Biomechanics

The function of the SIJ is to dissipate loads of the torso through the pelvis to the lower extremities and vice versa. The pelvis acts as a central base through which large forces are accepted and dissipated. Although the main role of the joint is to provide stability, the SIJ has limited motion that allows it to dissipate and transfer significant loads and stresses. Studies by Weisel indicate that most movement occurs when rising from the sitting to the standing position. However, the amount of motion is small, making assessment of sacroiliac motion during physical examination quite difficult. Selvik suggested that hyperextension produces the greatest degree of motion (2° on average, with only minimal translation of 0.5-1.6 mm).

If the motion in the pelvis is asymmetric, then dysfunction can occur. Some conditions that cause asymmetric motion include leg-length inequalities, a unilaterally weak lower limb (eg, polio), tight myofascial structures (eg, iliopsoas), and scoliosis. Hip osteoarthritis can lead to leg-length shortening and SIJ pain.

Women may be at increased risk for SIJ problems because their broader pelvises, greater femoral neck anteversion, and shorter limb lengths lead to different, possibly predisposing, biomechanics. In addition, pregnancy often leads to stretching of the pelvis, specifically targeting the sacroiliac ligaments and possibly leading to dysfunction, hypermobility syndromes, and chronic pain.

Innervation

The nerve supply of the SIJ originates from multiple lumbosacral root levels with partial innervation from L2 (anterior joint) to S3 (posterior joint). Because the root innervation can vary so widely, the pain referral patterns from primary sacroiliac pain can also vary. Fortin et al interviewed multiple patients documented to have sacroiliac pain by anesthetizing the joint with lidocaine injections under fluoroscopic guidance.[8, 9] He found referral patterns ranging from localized buttocks pain to frank radicular leg pain and many other descriptions in between.

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Acromioclavicular Joint Injury

Background

Injuries in and around the shoulder are common in today's athletic society. Proper knowledge of the different problems and treatment options for shoulder disorders is necessary to get patients back to their preinjury state.

Acromioclavicular (AC) joint injuries are common and often seen after bicycle wrecks, contact sports, and car accidents. The acromioclavicular joint is located at the top of the shoulder where the acromion process and the clavicle meet to form a joint. Several ligaments surround this joint, and depending on the severity of the injury, a person may tear one or all of the ligaments. Torn ligaments lead to acromioclavicular joint sprains and separations.[1]

The distal clavicle and acromion process can also be fractured. Injury to the acromioclavicular joint may injure the cartilage within the joint and can later cause arthritis of the acromioclavicular joint.

This article discusses the anatomy of the acromioclavicular joint, the diagnosis of disorders of this joint, and the different treatment options.

For excellent patient education resources, see eMedicineHealth's First Aid & Injuries Center. Also, see eMedicineHealth's article on Shoulder Dislocation.

NextEpidemiologyFrequencyUnited States

Injuries to the acromioclavicular joint are the most common reason that athletes seek medical attention following an acute shoulder injury. Glenohumeral dislocations (see Shoulder Dislocation) are the second most common injuries seen. Men in their second through fourth decades of life have the greatest frequency of acromioclavicular joint injuries, which are most often incomplete tears of the ligaments.[1]

PreviousNextFunctional Anatomy

The normal width of the acromioclavicula joint is 1-3 mm in younger individuals; it narrows to 0.5 mm or less in individuals older than 60 years.

The acromioclavicular joint is made up of 2 bones (the clavicle and the acromion), 4 ligaments, and a meniscus inside the joint.

The acromioclavicular joint is surrounded by a thin joint capsule and 4 small ligaments. These ligaments mostly give joint stability to anterior and posterior translation, as well as provide horizontal stability to the joint.

Another set of ligaments also provides vertical stability to the acromioclavicular joint. These ligaments are called the coracoclavicular ligaments, which are found medial to the acromioclavicular joint and go from the coracoid process on the scapula to the clavicle.

Different injuries result in different tears of the 2 coracoclavicular ligaments (the conoid and the trapezoid). Torn acromioclavicular joint ligaments and/or torn coracoclavicular ligaments are seen in acromioclavicular joint sprains. The meniscus that lies in the joint may also be injured during sprains or fractures around the acromioclavicular joint. The acromioclavicular capsular ligaments provide most of the joint stability in the anteroposterior (AP) direction. The conoid and trapezoid ligaments aid in providing superior-inferior stability to the joint. Compression of the joint is restrained mainly by the trapezoid ligament.

PreviousNextSport-Specific Biomechanics

When a person falls onto their shoulder, the force pushes the tip of the shoulder down. The clavicle is usually kept in its anatomic position, whereas the shoulder is driven down, which injures the different ligaments or causes a fracture. When the ligaments are injured they are either sprained or, in more severe cases, torn.

Acromioclavicular joint sprains have been classified according to their severity. In a type I sprain, a mild force applied to these ligaments does not tear them. The injury simply results in a sprain, which hurts, but the shoulder does not show any gross evidence of an acromioclavicular joint dislocation. Type II sprains are seen when a heavier force is applied to the shoulder, disrupting the acromioclavicular ligaments but leaving the coracoclavicular ligaments intact. When these injuries occur, the lateral clavicle becomes a little more prominent.

In type III sprains, the force completely disrupts the acromioclavicular and coracoclavicular ligaments. This leads to complete separation of the clavicle and obvious changes in appearance. The lateral clavicle is very prominent. A few more types of acromioclavicular joint sprains have been classified, but types I–III are the most common (see below).

Classification of acromioclavicular joint injuries.

An acromioclavicular joint sprain is more common than a fracture after an injury. However, fractures of the distal clavicle and the acromion process may occur, so the healthcare provider must be aware of such injuries and ready to diagnose and treat them as well (see Clavicular Injuries).

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Patellofemoral Joint Syndromes

Background

Patellofemoral joint complaints are one of the most common musculoskeletal complaints in all age groups. Complaints vary from anterior knee pain to peripatellar knee pain to retropatellar knee pain.[1, 2, 3, 4, 5, 6, 7] Nonspecific complaints may include global or generalized knee pain, joint line pain, or posterior knee pain. Often, there is a paucity of objective findings despite subjective complaints. The problem may vary from one of short duration to one of a recurrent or chronic nature.

The etiology of patellofemoral joint syndrome is multifactorial and results from a combination of intrinsic and extrinsic factors.[1, 2, 3, 4, 5, 6, 7, 8] Treatment is often conservative in nature. Because of the variable nature of the complaints and an often lack of objective identifiable pathologic cause of patellofemoral joint complaints, this condition can be difficult to evaluate, diagnose, and treat, which may cause great frustration for the physician and patient alike.[5]

For excellent patient education resources, visit eMedicineHealth's First Aid and Injuries Center and Osteoporosis Center. Also, see eMedicineHealth's patient education articles Knee Pain and Knee Injury.

Related Medscape Reference topics:

Patellofemoral Arthritis

Plica Syndrome

NextEpidemiologyFrequencyUnited States

Patellofemoral joint syndrome may affect as many as 25% of all athletes.

PreviousNextFunctional Anatomy

The patellofemoral joint is composed of the articulation of the patella with the femoral condyles of the femur. The patella has a configuration of a triangle with its apex directed inferiorly. Superiorly, it articulates with the trochlea, the distal articulating surface of the femur.

The patella is the largest sesamoid bone in the body and protects the knee from direct trauma. Localized within the quadriceps tendon, the patella also acts as a fulcrum for extension of the quadriceps.

Medial movement of the patella is controlled by the vastus medialis oblique (VMO) muscle. Lateral tracking is guided by both the vastus lateralis and the iliotibial band. Patellar motion is further constrained by the patellofemoral ligament, the patellotibial ligament, and the retinaculum.

The patella is engaged with the trochlea at 20-30 º of knee flexion. At 90 º, the patella contacts the lateral and medial femoral facets within the condylar fossa. At 130-135 º of knee flexion, the medial facets of the patella contact the articulating surface of the femoral condyles. In knee extension, the patella abuts the suprapatellar fat pad.

PreviousNextSport-Specific Biomechanics

The patella lies within the quadriceps tendon and thereby increases the mechanical advantage of the quadriceps mechanism. Not only does the patella increase the force of knee extension by 50%, but it also provides stability to the patellar tendon and minimizes the forces placed on the femoral condyles.

Tracking of the patella begins with the lower patellar border lying in contact with the suprapatellar fat pad when the knee is fully extended. With knee flexion, the patella moves proximally with a lateral shift, which is limited in excursion by the lateral retinaculum. As the knee continues to flex, the tibia internally rotates and the patella moves upward. The amount of force placed on the patellofemoral joint increases with increasing knee flexion. On the other hand, knee hyperflexion increases patellofemoral stress, as does extreme extension.

The vector force placed on the patella may be affected by the Q-angle.[9] The Q-angle is a line created from the anterior superior iliac spine (ASIS) to the mid patella, which intersects with a line from the mid patella to the tibial tubercle when the knee is in full extension. An average Q-angle for a male is 14 º, whereas that for a female is 17 º. Q-angles larger than average can indicate abnormal patellar tracking.

Other factors that may affect the vector force on the patella include the following:

Femoral anteversionTibial torsionHyperpronation of the footAtrophy of the VMO muscleA tight lateral retinaculumPatella position (patella alta/baja or subluxation)Inflexibility of the quadriceps, hamstring, iliotibial, and calf muscle-tendon unitsGeneral ligamentous laxityPreviousProceed to Clinical Presentation , Patellofemoral Joint Syndromes

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Metacarpophalangeal Joint Dislocation

Background

Sprains and dislocations of the metacarpophalangeal (MCP) joint of the finger are relatively rare due to the protected position of this joint in the hand.[1, 2, 3, 4] Injuries to the MCP joint of the thumb are more common, although these usually consist of collateral ligament injuries rather than dorsal or palmar dislocations.[5, 6]

MCP joint dislocation is seen in the image below.

Metacarpophalangeal joint dislocation of the small finger. Posteroanterior radiograph demonstrates loss of joint space.

For patient education resources, see the Bone, Joint, and Muscle Center and Breaks, Fractures, and Dislocations Center, as well as Finger Dislocation, Broken Finger, and Sprains and Strains.

NextFunctional Anatomy

The bony anatomy of the finger MCP joint provides greater laxity in extension, with the shallow articular surface of the proximal phalanx resting on the spherical metacarpal head. The metacarpal head is wider in palmar orientation, which leads to increasing bony stability as the joint approaches maximal flexion. Soft-tissue constraints, including the volar plate, accessory and true collateral ligaments, dorsal capsule, extensor tendon and sagittal band, and intrinsic tendons provide additional stability to the MCP joint. This results in an arc of motion from 30º of hyperextension to 120º of flexion, 30-40º of mediolateral laxity, and a small degree of rotational laxity.

The volar plate is a fibrocartilaginous structure firmly attached to the base of the proximal phalanx. Its origin, just proximal to the metacarpal head, is thin and diaphanous; this allows hyperextension of the MCP joint, but it is also the part of the joint most susceptible to injury during dislocations. The deep transverse metacarpal ligaments further stabilize the volar plates of the neighboring MCP joint.

The collateral ligaments originate from mediolateral depressions in the metacarpal head and travel in a distal-palmar direction to insert onto the base of the proximal phalanx. The elliptical shape of the metacarpal head causes these ligaments to loosen in extension and tighten in flexion. The accessory collateral ligament spans from the true collateral ligament to the volar plate, providing additional joint stability in extension. The central extensor tendon and sagittal band augment the thin dorsal capsule. The tendons of the palmar and dorsal interossei add a small degree of dynamic stability.

The MCP joint of the thumb is a condyloid (hinged) joint, with a quadrilateral rather than spherical metacarpal head. The capsule and ligaments of this joint are similar to those of the finger MCP joint. Additionally, the volar plate of the thumb MPJ usually contains 2 sesamoids that articulate with the metacarpal head. The insertion of the thenar muscles into the sesamoids contributes to joint stability. These bony and ligamentous constraints allow less motion than in the MCP joint of the fingers, especially in lateral motion and rotation; abduction and adduction average 10º and a slight amount of pronation occurs during flexion.

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Dorsal MCP joint dislocations have been described as simple or complex. Simple dislocations are those in which no soft tissue is interposed in the joint. These are usually reduced easily with an appropriate closed technique. In a classic article published in 1957, Kaplan elegantly described the anatomic features of the complex MCP joint dislocation.[7] A metacarpal head displaced in palmar orientation sits between the lumbrical muscle radially and the flexor tendons ulnarly. The volar plate, still firmly attached to the base of the proximal phalanx, is displaced into the MCP joint. Longitudinal traction only further tightens these already taut soft tissues, trapping the metacarpal head. Complex dislocations usually require open reduction.

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